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Fig. 4 | BMC Cell Biology

Fig. 4

From: Microvesicles derived from hypoxia/reoxygenation-treated human umbilical vein endothelial cells promote apoptosis and oxidative stress in H9c2 cardiomyocytes

Fig. 4

H/R-EMVs induced the oxidative damage in H9c2 cells. a The activity of SOD in H9c2 cells was examined after being treated with 10, 30, 60 μg/mL H/R-EMVs for 6 h. b MDA content in H9c2 cells were determined after being treated with 10, 30, 60 μg/mL H/R-EMVs for 6 h. c, d Representative images and flow cytometry analysis of ROS production in H9c2 cells after H/R-EMVs treatment. ** p < 0.01, *** p < 0.001 versus Control; # p < 0.05, ## p < 0.01, ### p < 0.001 versus H/R-EMVs 10 μg/mL; &&& p < 0.001 versus H/R-EMVs 30 μg/mL

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