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Fig. 5 | BMC Molecular and Cell Biology

Fig. 5

From: The primary cilium dampens proliferative signaling and represses a G2/M transcriptional network in quiescent myoblasts

Fig. 5

A working model for primary cilium function in myoblast quiescence.

Control myoblasts entering suspension induced quiescence elaborate primary cilia, which work to suppress proliferative signaling in G0, whereas myoblasts where ciliogenesis is blocked enter an altered quiescent state characterized by arrest in G2 and activation of G2/M checkpoint genes.

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